The complex pathophysiologic mechanisms of PCOS involve multiple endocrine disorders and metabolic disturbances
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FSP1 can inhibit ferroptosis by reducing ubiquinone or CoQ to ubiquinone or CoQH 2 on the cell membrane, with the latter functioning as an antioxidant that trap radicals and thus prevents lipid peroxidation, thereby preventing ferroptosis (Fig
Since the liver, as an organ directly involved in many physiological processes including bilirubin metabolism processes, has a more complex relationship with bilirubin, the details of which will be described later, the sections other than hepatocellular carcinoma (HCC) are described here